The long-term prognosis of subendocardial myocardial infarction by William Kramer Levy

Cover of: The long-term prognosis of subendocardial myocardial infarction | William Kramer Levy

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  • Myocardial infarction

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Pagination72 leaves ;
Number of Pages72
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Open LibraryOL26393118M

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Long-term prognosis after first Q-wave (transmural) or non-Q-wave (nontransmural) myocardial infarction: analysis of patients. Am J Cardiol. Aug; 52 (3)– Fabricius-Bjerre N, Munkvad M, Knudsen JB. Subendocardial and transmural myocardial infarction: a five year survival study.

Am J Med. Jun; 66 (6)–Cited by: Long-term prognosis of unrecognized myocardial infarction detected with cardiovascular magnetic resonance in an elderly population. study to investigate the relationship between CMR detected UMIs and cardiac death or other severe cardiac events in long-term follow-up Cited by:   Viewpoints concerning the course and prognosis of non-Q wave myocardial infarction (NQMI) have evolved considerably in the last two decades.

The NQMI or subendocardial myocardial infarction (SEMI) was once thought to be a relatively benign condition with an uncomplicated course and good prognosis.

1 This idea has since been challenged. Two recent editorials2, 3 emphasize the Cited by: 2. Many patients with nontransmural myocardial infarction suffer from angina pectoris after the infarction [4].

Thus the long-term prognosis remains controversial [1, 2, 3, 5].Cited by: 2. International Journal of Cardiology, 24 () Elsevier CARDIO Short-term and long-term prognosis after myocardial infarction: prognostic value of coronary anatomy and left ventriculography C.C. de Cock, F.C. Visser, M.J.

van Eenige and J.P. Roos Department of Cardiology, Free University Hospital, Amsterdam, The Netherlands (Received 23 September ; Cited by: 4. Of the 3 patients who had documented coronary artery disease and a cardiac event, their LGE pattern was: 1 subendocardial pattern typical for previous myocardial infarction, 1 focal subendocardial pattern atypical for previous myocardial infarction, and 1 subepicardial pattern atypical for previous myocardial infarction.

the isolated non-transmural or subendocardial infarction - has only been recognized as a distinct clinical entity relatively recently. Barnes and Ball''' reported the autopsy findings of 49 patients with The long-term prognosis of subendocardial myocardial infarction book infarction and noted that three of these patients had diffuse involvement.

of the subendocardial area without damage in the outer. Cannom DS, Levy W, Cohen LS. The short- and long-term prognosis of patients with transmural and nontransmural myocardial infarction.

Am J Med. Oct; 61 (4)– Szklo M, Goldberg R, Kennedy HL, Tonascia JA. Survival of patients with nontransmural myocardial infarction: a population-based study. Am J Cardiol. Oct; 42 (4)–   Previous reports1, 2, 3 have described the details of the coronary heart disease study among residents of Rochester, Minnesota, including the method of diagnosis of angina pectoris and myocardial infarction.

4 This particular report is focused on the hospital and posthospital course of patients who have had transmural or subendocardial myocardial infarction. Objective To evaluate the prognosis of unrecognised myocardial infarction determined by electrocardiography (UMI-ECG) or cardiac magnetic resonance imaging (UMI-CMR).

Design Systematic review and meta-analysis of prospective studies. Data sources Electronic databases, including PubMed, Embase, and Google Scholar.

Study selection Prospective cohort studies were. Waris EK, Siitonen L, Himanka E. Heart size and prognosis in myocardial infarction. Am Heart J.

Feb; 71 (2)– Kentala E, Pyörälä K, Heikkilä J, Sarna S, Luurila O. Factors related to long-term prognosis following acute myocardial infarction.

Importance of left ventricular function. Scand J Rehabil Med. ; 7 (3)– Furthermore nine of them had attacks of non-Q-wave myocardial infarction with severe ST segment depression in many leads.

At necropsy five of six patients who had shown severe ST segment depressions in many leads at the onset of non-Q-wave myocardial infarction were found to have circumferential subendocardial lesions with triple vessel disease. Myocardial infarction is characterized by necrosis resulting from an insufficient supply of oxygenated blood to an area of the heart.

According to the joint European Society of Cardiology/American College of Cardiology, either one of the following criteria for acute evolving or recent MI satisfies the diagnosis: a. Survival of Patients With Nontransmural Myocardial Infarction: A Population-Based Study MOYSES SZKLO, MD, Dr PH ROBERT GOLDBERG, MS, PhD HAROLD L.

KENNEDY, MPH, MD, FACC JAMES A. TONASCIA, PhD Baltimore, Maryland A population-based study was conducted in metropolitan Baltimore in which the short- and long-term prognosis of patients with non- transmural myocardial infarction. These data suggest that patients with either “subendocardial” or “transmural” infarctions can be subdivided into low and high risk categories regarding both hospital and long-term prognosis.

Patients with a normal QRS complex initially have the greatest likelihood of a benign prognosis following an acute myocardial infarction. The aim of the study was to assess the relationship between paroxysmal atrial fibrillation during acute myocardial infarction and the long-term prognosis of patients after acute myocardial infarction.

The incidence of paroxysmal a trial fibrillation among consecutive hospitalized patients. Introduction. Current management of acute myocardial infarction (MI) is based on a prompt diagnosis and immediate revascularization through a diagnostic coronary angiography and subsequent revascularization, mostly percutaneous interventions.

1 About 90% of patients presenting with ST-segment elevation MI have an explanatory coronary artery stenosis or occlusion. 2 For these. Introduction. Incomplete recovery of myocardial tissue perfusion despite successful opening of the culprit artery by primary percutaneous coronary intervention (PPCI) occurs in approximately half of patients with ST-elevation myocardial infarction (STEMI).

1, 2 The two major underlying pathologies in this complex process mainly caused by ischaemia–reperfusion injury are microvascular. John B. Kostis's research works w citations and 7, reads, including: Readmission and mortality among heart failure patients with history of hypertension in a statewide database.

Acute Myocardial Infarction Patrick J Gallagher MD PhD transmural and subendocardial infarcts. Incidence of Acute Myocardial Infarction •In the short and long term the pattern of cardiac pathology is very different to conventional textbook descriptions.

Abstract. During the last 30 years, it has been consistently observed that patients with non-Q-wave myocardial infarction (MI) have a greater likelihood of subsequent fatal and non-fatal cardiac events, particularly during long-term follow-up, than do patients with Q-wave MI.

Acute myocardial infarction (MI) can occur from increased myocardial oxygen demand and/or reduced supply in the absence of acute atherothrombotic plaque disruption; a condition called type 2 myocardial infarction (T2MI).

As with any MI subtype, there must be clinical evidence of myocardial ischemia to make the diagnosis. This condition is increasingly diagnosed due to the increasing. A study was carried out in metropolitan Baltimore in which the short- and long-term prognosis of patients with anterior myocardial infarction (MI) was compared with that of patients with.

Acute myocardial infarction is myocardial necrosis resulting from acute obstruction of a coronary artery. Symptoms include chest discomfort with or without dyspnea, nausea, and diaphoresis. Diagnosis is by ECG and the presence or absence of serologic markers. Myocardial infarction (MI) or acute myocardial infarction (AMI) is the death of heart muscle from the sudden blockage of a coronary artery by a blood clot.

Myocardial infarction most commonly due to occlusion (blockage) of a coronary artery following the rupture of a vulnerable atherosclerotic plaque, which is an unstable collection of lipids (cholesterol and fatty acids) and white.

Acute myocardial infarction is myocardial necrosis resulting from acute obstruction of a coronary artery. Symptoms include chest discomfort with or without dyspnea, nausea, and diaphoresis. Diagnosis is by ECG and the presence or absence of serologic markers.

Treatment is antiplatelet drugs. A patient who has a prior subendocardial myocardial infarction is admitted with an impending acute myocardial infarction • The physician ’ s final diagnosis is “ acute subendocardial myocardial infarction ” • ICDCM = ; POA = Y • This acute condition is present on admission.

Twenty-eight patients who were two weeks post acute myocardial infarction walked on a motorized treadmill at 1 MPH 0 percent grade for five minutes (group 1) or to an end-point of symptoms, ST-T wave changes or arrhythmias (group 2).

At subsequent cardiac catheterization, 73 percent in group 1 had single vessel coronary artery disease whereas 82 percent group 2 had three or four vessel.

Patients with myocardial infarction resulting from acute coronary syndrome are classified by electrocardiographic presentation: 1-acute ST-segment elevation myocardial infarction (STEMI) or 2-non-ST-segment elevation myocardial infarction (NSTEMI). Prompt reperfusion of an infarct-related artery by percutaneous coronary interventions provides some relief of symptoms; long-term prognosis.

Acute myocardial infarction results from the cessation of myocardial blood flow caused by thrombotic occlusion of a coronary artery. Rapid restoration of blood flow to the ischemic myocardium minimizes cardiac damage and improves early and long-term morbidity and mortality.

Chest pain is the first symptom of myocardial infarction, but in some patients with silent ischemia, the disease can be. Acute Subendocardial Myocardial Infarction in Patients Its Detection by Technetium m Stannous Pyrophosphate Myocardial Scintigrams By JAMES T.

WILLERSON, M.D., ROBERT W. PARKEY, M.D., FREDERICK J. BONTE, M.D., STEVEN L. MEYER, M.D., AND ERNEST M. STOKELY, PH.D. SUMMARY Eighty-eight patients admitted to a coronary care unit with chest pain of varying etiology.

Background and Objectives: Thyroid hormones (TH) affect cardiac function through effects on cardiac contractility and systemic vascular resistance. While TH replacement for patients with hypothyroidism might be necessary for restoration of cardiac output after an acute myocardial infarction (AMI), it could theoretically lead to excessively rapid restoration of the metabolic rate.

Myocardial infarction (MI) refers to tissue death of the heart muscle caused by ischaemia, that is lack of oxygen delivery to myocardial is a type of acute coronary syndrome, which describes a sudden or short-term change in symptoms related to blood flow to the heart.

Unlike the other type of acute coronary syndrome, unstable angina, a myocardial infarction occurs when there is cell. is a rapid access, point-of-care medical reference for primary care and emergency clinicians.

Started inthis collection now contains interlinked topic pages divided into a tree of 31 specialty books and chapters. Acute myocardial infarction is the medical name for a heart attack. A heart attack is a life-threatening condition that occurs when blood flow to the heart muscle is abruptly cut off, causing.

Szyma?ski P, Rezler J, Stec S, Budaj A. Long-term prognostic value of an index of myocardial performance in patients with myocardial Cardiol ; Bruch C, Schmermund A, Dagres N, Katz M, Bartel T, Erbel R. Tei-Index in coronary artery disease-validation in patients with overall cardiac and isolated diastolic dysfunction.

• Subendocardial infarct: involves inner 1/3 or upto half • Post myocardial infarction syndrome (Dresslers synd): in %, after weeks of MI, pneumonitis. Prognosis • Long term prognosis depends on ventricular function and degree of coronary obstruction. He has an acute myocardial infarction and is admitted into the critical coronary care unit.

Five days later while still in the hospital, he experiences another acute myocardial infarction. The final diagnoses are acute transmural myocardial infarction of inferior wall, subsequent acute subendocardial myocardial infarction, and acute hypotension.

Long-term life expectancy for acute myocardial infarction and heart failure patients after hospitalization in Ontario, Canada by Dennis T. Ko, edition, in English. Background: In the era of fibrinolysis, women suffered from higher early and late mortality rates than men after acute ST-elevation myocardial infarction (STEMI).

Primary percutaneous coronary intervention (PCI) has been determined to be the most effective therapy strategy in STEMI.

It is not clear if female gender is an independent predictor of a worse long-term prognosis among patients who.

The British Cardiac Society commissioned this report to help address inconsistencies in the terminology for acute coronary syndromes and wide variations in the threshold for the diagnosis of myocardial infarction (MI) depending on the assay performed, the precision, and the sensitivity.

In addition, several publications have highlighted potential problems with the application of the European. Furthermore, CMR can detect subendocardial myocardial infarction, missed by SPECT/PET. The CMR extent of scar predicts the potential for functional recovery after revascularisation.

36–38 Download original Subendocardial and/or transmural LGE, following the distribution of coronary arteries, are indicative of CAD.Objective To evaluate the prognosis of unrecognised myocardial infarction determined by electrocardiography (UMI-ECG) or cardiac magnetic resonance imaging .

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